Teachers who used transcendental meditation (TM) for four months had significant reductions in burnout, stress, fatigue, and depression, with increases in resilience.
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Dysfunction in anti-inflammatory cells called MRC1+ macrophagesĀ are implicated in neuropathic pain. Stimulating the expression of these cells using gene therapy could potentially treatĀ chronic pain.
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Changes to the transcriptome, or the full set of messenger RNA molecules that synthesize proteins from genetic material, are linked to biological aging.
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In addition to helping to prevent Alzheimer's disease, aerobic exercise may also slow down memory loss in people already diagnosed with Alzheimer's.
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Curcumin suppresses the aggregation of amyloid proteins that accumulate in Alzheimer's disease. In mice, curcumin also improved fat and cholesterol metabolism.
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Aging increases the risk of lung fibrotic disorders; the sirtuin SIRT3 is reduced in these disorders. Boosting SIRT3 reversed the cell dysfunction that causes lung fibrosis, improving the health of aged mice.
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More than one third of genetic variants that increase the risk of coronary artery diseaseĀ are located in the liver, regulating the expression of genes related to glucose and cholesterol metabolism.
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New compound eliminates the degeneration of upper motor neurons that contribute to ALS (Lou Gehrig's disease). The compound, NU-9, reduces protein misfolding and clumping.
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The gene FOXO3 is associated with longevity and humans living past age 100. New research finds that FOXO3 protects the brain's stem cells from stress-induced damage, preserving its ability to regenerate.
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Estrogen deficiency during menopause changes microRNA signaling in skeletal muscle, activating a series of cell-death signaling cascades that are detrimental to muscle mass.
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Biodegradable microcapsules encourage the development of new neurons in the hippocampus of the brain by delivering nerve growth factor. If applied to humans, this could reverse many cases of neurodegeneration.
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Skeletal muscle plays a role in controlling glucose levels, which is vital for reducing the risk of metabolic diseases like type 2 diabetes. More skeletal muscle has increased activity of a gene called (KLF15).
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